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rabbit anti map2 polyclonal  (Proteintech)


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    Structured Review

    Proteintech rabbit anti map2 polyclonal
    Rabbit Anti Map2 Polyclonal, supplied by Proteintech, used in various techniques. Bioz Stars score: 96/100, based on 465 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/rabbit+anti+map2/MAP2+Antibody/pmc13045046-1-0-4
    Average 96 stars, based on 465 article reviews
    rabbit anti map2 polyclonal - by Bioz Stars, 2026-10
    96/100 stars

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    Article Title: A modular platform for automated organoid culture and longitudinal imaging.
    Article Snippet: The primary antibodies used were mouse and rabbit anti-Map2 (Proteintech # 17490- 1-AP, 1:2,000).

    Article Title: GABA-dependent microglial elimination of inhibitory synapses underlies neuronal hyperexcitability in epilepsy.
    Article Snippet: Neuronal hyperexcitability is a common pathophysiological feature of many neurological diseases.. Neuron–glia interactions underlie this process but the detailed mechanisms remain unclear.. Here, we reveal a critical role of microglia-mediated selective elimination of inhibitory synapses in driving neuronal hyperexcitability.

    Article Title: A modular platform for automated organoid culture and longitudinal imaging
    Article Snippet: The primary antibodies used were mouse and rabbit anti-Map2 (Proteintech # 17490-1-AP, 1:2,000).

    Incubation:

    Article Title: Endothelial cells temporally orchestrate lymphocyte dynamics and functional outcomes after ischemic stroke.
    Article Snippet: .. Then, the sections were incubated at room temperature for 1 hour with primary antibodies as follows: Rabbit anti-MAP2 (Proteintech, 17490-1-AP, 1:200), Rabbit anti-CD19 (Abcam, ab245235, 1:100), Rabbit anti-CD3e (Abcam, ab16669, 1:100), Rabbit anti-NKp46 (Abcam, ab233558, 1:200), Rabbit anti-C1q (Abcam, ab182451, 1:100), Rat anti-TREM2 (Abcam, ab86491, 1:50), Rabbit anti-CSF1R (Abcam, ab254357, 1:100), Rabbit anti-CX3CR1 (Abcam, ab8020, 1:100). ..

    Article Title: Skin-Derived Precursor Cell-Differentiated Dopaminergic Neurons Promote Functional Recovery in Parkinson’s Disease via Tunneling Nanotube-Mediated Intercellular Communication
    Article Snippet: .. The cells were Page 15/33 incubated overnight at 4°C with the following primary antibodies: rabbit anti-TUBB3 (Zenbio, R23620), rabbit anti-MAP2 (Proteintech, 67015-1-Ig), rabbit anti-SYN (Proteintech, 10842-1-AP), and mouse antiNEUN (Abcam, Ab104224). .. The antibodies utilized included rabbit anti-TH (Abcam, Ab104224), rabbit anti-TH (Abcam, Ab137869), chicken anti-TH (Abcam, Ab76442), rabbit anti-GABA (Servicebio, GB114791), rabbit anti-vGlut1 (Servicebio, GB11821), goat anti-CHAT (Servicebio, GB11070), rabbit antiDAT (Proteintech, 22524-1-AP), rabbit anti-NURR1 (Proteintech, 10975-2-AP), rabbit anti-PAX6 (Servicebio, GB11777), rabbit anti-SOX2 (Abcam, Ab92494), rabbit anti-NESTIN (Abcam, Ab105389), rabbit anti-FOX2 (CST, 8186), and mouse anti-GFAP (Servicebio, GB12100) antibodies.

    Article Title: The expression profile of type 1 equilibrative nucleoside transporter in aged C57BL/6J mouse brain.
    Article Snippet: .. The sections were deparaffinized, rehydrated, subjected to antigen repair, blocked and washed, and then incubated with the first primary antibodies including rabbit antiIba1 (10904-1-AP, 1:500; Proteintech, Wuhan, China), mouse antiGFAP (38014, 1:100; SAB, Greenbelt, USA), rabbit anti-MAP2 (17490-1-AP, 1:500; Proteintech), rabbit anti-PSD95 (20665-1-AP, 1:200; Proteintech), mouse anti-MBP (83683S, 1:500; CST, Danvers, USA), rabbit anti-AChE (AF5274, 1:100; Affinity Biosciences, Cincinnati, USA), rabbit anti-DAT (DF4529, 1:100; Affinity Biosciences), rabbit anti-VGluT1 (DF13657, 1:100; Affinity Biosciences), rabbit anti-GAD65 (21760-1-AP, 1:500; Proteintech), rabbit antiGM130 (A11408, 1:500; ABclonal, Wuhan, China), rabbit antiCalnexin (A15631, 1:500; ABclonal), rabbit anti-LAMP1 (ab278043, 1:100; Abcam, Cambridge, UK), rabbit anti-TOMM20 (ET1609-25, 1:100; HUABIO, Hangzhou, China), and rabbit anti-COX IV (AF5468, 1:100; Affinity Biosciences) overnight at 4°C, After washing with PBST, the sections were incubated with the first HRP-conjugated secondary antibody for 30 min at room temperature, followed by TYR-520 (excitation light 490 nm) incubation for approximately 3–10 min. .. The sections were then subjected to repeated antigen repair, blocking and washing, and incubated with the second primary antibody rabbit anti-ENT1 (YT5031, 1:100; Immunoway) overnight at 4°C.

    Article Title: Repair-associated macrophages increase after early-phase microglia attenuation to promote ischemic stroke recovery
    Article Snippet: .. They were then blocked with 5% normal donkey serum in 0.3% PBST at room temperature for 1 h before overnight incubation at 4 °C with the following primary antibodies: Goat anti-CD31 (R&D, AF3628, 1:200), Rabbit anti-ZO1 (Abcam, ab221547, 1:200), Rabbit anti-Pdgfra (Abcam, ab203491, 1:250), Goat anti-IBA1 (Abcam, ab5076, 1:250), Mouse anti-APC (Sigma-Aldrich, OP80, 1:100), Rat anti-BrdU (Abcam, ab6326, 1:100), Mouse anti-SMI32 (BioLegend, 801701, 1:250), Rat anti-MBP (Abcam, ab7349, 1:250), Rabbit anti-Plin2 (Abcam, ab108323, 1:100), Rabbit anti-GPNMB (Abcam, ab188222, 1:200), Rabbit anti-CD63 (Abcam, ab217345, 1:200) and Rabbit anti-MAP2 (Proteintech, 17490-1-AP, 1:500). .. After washing with 0.3% PBST three times for 10 minutes each, brain slices were incubated with appropriate secondary antibodies conjugated with Donkey anti-Goat Alexa Fluor 488 (Invitrogen, 1:500, A-11055), Donkey anti-Rabbit Alexa Fluor 488 (Invitrogen, 1:500, A-21206), Donkey anti-Mouse Alexa Fluor 488 (Invitrogen, 1:500, A-21202), Donkey anti-Rat Alexa Fluor 488 (Invitrogen, 1:500, A-21208), Goat anti-Rabbit Alexa Fluor 555 (Invitrogen, 1:500, A-21428), Donkey anti-Rabbit Alexa Fluor 594 (Invitrogen, 1:500, A-21207), Donkey anti-Goat Alexa Fluor 594 (Invitrogen, 1:500, A-11058), Donkey anti-Rat Alexa Fluor 594 (Invitrogen, 1:500, A-21209), Goat anti-Mouse Alexa Fluor 594 (Invitrogen, 1:500, A-11005), Donkey anti-Goat Alexa Fluor 647 (Invitrogen, 1:500, A-21447), Donkey anti-Rabbit Alexa Fluor 647 (Invitrogen, 1:500, A-31573) in a dark environment at room temperature for 1 h. Subsequently, brain slices were washed with PBS three times and mounted on glass slides with mount-G containing DAPI (Yeasan Biotech).

    Article Title: Repair-associated macrophages increase after early-phase microglia attenuation to promote ischemic stroke recovery.
    Article Snippet: .. They were then blocked with 5% normal donkey serum in 0.3% PBST at room temperature for 1 h before overnight incubation at 4 °C with the following primary antibodies: Goat anti-CD31 (R&D, AF3628, 1:200), Rabbit anti-ZO1 (Abcam, ab221547, 1:200), Rabbit anti-Pdgfra (Abcam, ab203491, 1:250), Goat anti-IBA1 (Abcam, ab5076, 1:250), Mouse anti-APC (Sigma-Aldrich, OP80, 1:100), Rat anti-BrdU (Abcam, ab6326, 1:100), Mouse anti-SMI32 (BioLegend, 801701, 1:250), Rat anti-MBP (Abcam, ab7349, 1:250), Rabbit anti-Plin2 (Abcam, ab108323, 1:100), Rabbit anti-GPNMB (Abcam, ab188222, 1:200), Rabbit anti-CD63 (Abcam, ab217345, 1:200) and Rabbit anti-MAP2 (Proteintech, 17490-1-AP, 1:500). .. After washing with 0.3% PBST three times for 10minutes each, brain slices were incubated with appropriate secondary antibodies conjugated with Donkey anti-Goat Alexa Fluor 488 (Invitrogen, 1:500, A-11055), Donkey antiRabbit Alexa Fluor 488 (Invitrogen, 1:500, A-21206), Donkey antiMouse Alexa Fluor 488 (Invitrogen, 1:500, A-21202), Donkey anti-Rat Alexa Fluor 488 (Invitrogen, 1:500, A-21208), Goat anti-Rabbit Alexa Fluor 555 (Invitrogen, 1:500, A-21428), Donkey anti-Rabbit Alexa Fluor 594 (Invitrogen, 1:500, A-21207), Donkey anti-Goat Alexa Fluor 594 (Invitrogen, 1:500, A-11058), Donkey anti-Rat Alexa Fluor 594 (Invitrogen, 1:500, A-21209), Goat anti-Mouse Alexa Fluor 594 (Invitrogen, 1:500, A-11005), Donkey anti-Goat Alexa Fluor 647 (Invitrogen, 1:500, A-21447), Donkey anti-Rabbit Alexa Fluor 647 (Invitrogen, 1:500, A-31573) in a dark environment at room temperature for 1 h. Subsequently, brain slices were washed with PBS three times and mounted on glass slides with mount-G containing DAPI (Yeasan Biotech).



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    Waveform rescue and impairment of AIS in C9-MSNs (A) Example AP waveforms for Con-1 (blue) and C9-3 MSNs (black) before and after the addition of BK channel activator NS11021 (10 μM, lighter color shade). Scale bars, 20 mV, 10 ms. (B and C) Mean ± SEM AP amplitude, AHP, half-width, threshold, and rheobase of C9-3 MSNs in the presence and absence of NS11021, along with Con-1 MSNs, were examined in parallel. Statistics, ∗ p < 0.05 and ∗∗∗ p < 0.001, from paired t test or t test . Data: con-1: n = 8, N = 3. C9-3: n = 10, N = 4. (D) NS11021 treatment had no effect on AP output in C9-3 MSNs evoked from current stimulation. Con-1 patched in parallel, shown for reference. Data: Con-1: n = 8, N = 3. C9-3: n = 10, N = 4. Statistics, two-way repeated measures ANOVA followed by Tukey’s multiple comparisons test. (E) Images of Con-1 and C9-3 stained with AIS marker ankyrin-G, neuronal marker <t>MAP2,</t> and DAPI. Scale bars, 50 μm. (F) Mean ± SEM AIS length and distance of start of AIS from the cell soma. Data: Con-1: n = 16, N = 4. C9-3: n = 36, N = 4. ∗∗∗ p < 0.001, t test.
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    Wuhan Fine Biotech fitc conjugated rabbit anti mouse map2 primary antibody
    Waveform rescue and impairment of AIS in C9-MSNs (A) Example AP waveforms for Con-1 (blue) and C9-3 MSNs (black) before and after the addition of BK channel activator NS11021 (10 μM, lighter color shade). Scale bars, 20 mV, 10 ms. (B and C) Mean ± SEM AP amplitude, AHP, half-width, threshold, and rheobase of C9-3 MSNs in the presence and absence of NS11021, along with Con-1 MSNs, were examined in parallel. Statistics, ∗ p < 0.05 and ∗∗∗ p < 0.001, from paired t test or t test . Data: con-1: n = 8, N = 3. C9-3: n = 10, N = 4. (D) NS11021 treatment had no effect on AP output in C9-3 MSNs evoked from current stimulation. Con-1 patched in parallel, shown for reference. Data: Con-1: n = 8, N = 3. C9-3: n = 10, N = 4. Statistics, two-way repeated measures ANOVA followed by Tukey’s multiple comparisons test. (E) Images of Con-1 and C9-3 stained with AIS marker ankyrin-G, neuronal marker <t>MAP2,</t> and DAPI. Scale bars, 50 μm. (F) Mean ± SEM AIS length and distance of start of AIS from the cell soma. Data: Con-1: n = 16, N = 4. C9-3: n = 36, N = 4. ∗∗∗ p < 0.001, t test.
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    Novus Biologicals rabbit anti map2
    Waveform rescue and impairment of AIS in C9-MSNs (A) Example AP waveforms for Con-1 (blue) and C9-3 MSNs (black) before and after the addition of BK channel activator NS11021 (10 μM, lighter color shade). Scale bars, 20 mV, 10 ms. (B and C) Mean ± SEM AP amplitude, AHP, half-width, threshold, and rheobase of C9-3 MSNs in the presence and absence of NS11021, along with Con-1 MSNs, were examined in parallel. Statistics, ∗ p < 0.05 and ∗∗∗ p < 0.001, from paired t test or t test . Data: con-1: n = 8, N = 3. C9-3: n = 10, N = 4. (D) NS11021 treatment had no effect on AP output in C9-3 MSNs evoked from current stimulation. Con-1 patched in parallel, shown for reference. Data: Con-1: n = 8, N = 3. C9-3: n = 10, N = 4. Statistics, two-way repeated measures ANOVA followed by Tukey’s multiple comparisons test. (E) Images of Con-1 and C9-3 stained with AIS marker ankyrin-G, neuronal marker <t>MAP2,</t> and DAPI. Scale bars, 50 μm. (F) Mean ± SEM AIS length and distance of start of AIS from the cell soma. Data: Con-1: n = 16, N = 4. C9-3: n = 36, N = 4. ∗∗∗ p < 0.001, t test.
    Rabbit Anti Map2, supplied by Novus Biologicals, used in various techniques. Bioz Stars score: 93/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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    Waveform rescue and impairment of AIS in C9-MSNs (A) Example AP waveforms for Con-1 (blue) and C9-3 MSNs (black) before and after the addition of BK channel activator NS11021 (10 μM, lighter color shade). Scale bars, 20 mV, 10 ms. (B and C) Mean ± SEM AP amplitude, AHP, half-width, threshold, and rheobase of C9-3 MSNs in the presence and absence of NS11021, along with Con-1 MSNs, were examined in parallel. Statistics, ∗ p < 0.05 and ∗∗∗ p < 0.001, from paired t test or t test . Data: con-1: n = 8, N = 3. C9-3: n = 10, N = 4. (D) NS11021 treatment had no effect on AP output in C9-3 MSNs evoked from current stimulation. Con-1 patched in parallel, shown for reference. Data: Con-1: n = 8, N = 3. C9-3: n = 10, N = 4. Statistics, two-way repeated measures ANOVA followed by Tukey’s multiple comparisons test. (E) Images of Con-1 and C9-3 stained with AIS marker ankyrin-G, neuronal marker <t>MAP2,</t> and DAPI. Scale bars, 50 μm. (F) Mean ± SEM AIS length and distance of start of AIS from the cell soma. Data: Con-1: n = 16, N = 4. C9-3: n = 36, N = 4. ∗∗∗ p < 0.001, t test.
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    Waveform rescue and impairment of AIS in C9-MSNs (A) Example AP waveforms for Con-1 (blue) and C9-3 MSNs (black) before and after the addition of BK channel activator NS11021 (10 μM, lighter color shade). Scale bars, 20 mV, 10 ms. (B and C) Mean ± SEM AP amplitude, AHP, half-width, threshold, and rheobase of C9-3 MSNs in the presence and absence of NS11021, along with Con-1 MSNs, were examined in parallel. Statistics, ∗ p < 0.05 and ∗∗∗ p < 0.001, from paired t test or t test . Data: con-1: n = 8, N = 3. C9-3: n = 10, N = 4. (D) NS11021 treatment had no effect on AP output in C9-3 MSNs evoked from current stimulation. Con-1 patched in parallel, shown for reference. Data: Con-1: n = 8, N = 3. C9-3: n = 10, N = 4. Statistics, two-way repeated measures ANOVA followed by Tukey’s multiple comparisons test. (E) Images of Con-1 and C9-3 stained with AIS marker ankyrin-G, neuronal marker <t>MAP2,</t> and DAPI. Scale bars, 50 μm. (F) Mean ± SEM AIS length and distance of start of AIS from the cell soma. Data: Con-1: n = 16, N = 4. C9-3: n = 36, N = 4. ∗∗∗ p < 0.001, t test.
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    Waveform rescue and impairment of AIS in C9-MSNs (A) Example AP waveforms for Con-1 (blue) and C9-3 MSNs (black) before and after the addition of BK channel activator NS11021 (10 μM, lighter color shade). Scale bars, 20 mV, 10 ms. (B and C) Mean ± SEM AP amplitude, AHP, half-width, threshold, and rheobase of C9-3 MSNs in the presence and absence of NS11021, along with Con-1 MSNs, were examined in parallel. Statistics, ∗ p < 0.05 and ∗∗∗ p < 0.001, from paired t test or t test . Data: con-1: n = 8, N = 3. C9-3: n = 10, N = 4. (D) NS11021 treatment had no effect on AP output in C9-3 MSNs evoked from current stimulation. Con-1 patched in parallel, shown for reference. Data: Con-1: n = 8, N = 3. C9-3: n = 10, N = 4. Statistics, two-way repeated measures ANOVA followed by Tukey’s multiple comparisons test. (E) Images of Con-1 and C9-3 stained with AIS marker ankyrin-G, neuronal marker <t>MAP2,</t> and DAPI. Scale bars, 50 μm. (F) Mean ± SEM AIS length and distance of start of AIS from the cell soma. Data: Con-1: n = 16, N = 4. C9-3: n = 36, N = 4. ∗∗∗ p < 0.001, t test.
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    Waveform rescue and impairment of AIS in C9-MSNs (A) Example AP waveforms for Con-1 (blue) and C9-3 MSNs (black) before and after the addition of BK channel activator NS11021 (10 μM, lighter color shade). Scale bars, 20 mV, 10 ms. (B and C) Mean ± SEM AP amplitude, AHP, half-width, threshold, and rheobase of C9-3 MSNs in the presence and absence of NS11021, along with Con-1 MSNs, were examined in parallel. Statistics, ∗ p < 0.05 and ∗∗∗ p < 0.001, from paired t test or t test . Data: con-1: n = 8, N = 3. C9-3: n = 10, N = 4. (D) NS11021 treatment had no effect on AP output in C9-3 MSNs evoked from current stimulation. Con-1 patched in parallel, shown for reference. Data: Con-1: n = 8, N = 3. C9-3: n = 10, N = 4. Statistics, two-way repeated measures ANOVA followed by Tukey’s multiple comparisons test. (E) Images of Con-1 and C9-3 stained with AIS marker ankyrin-G, neuronal marker <t>MAP2,</t> and DAPI. Scale bars, 50 μm. (F) Mean ± SEM AIS length and distance of start of AIS from the cell soma. Data: Con-1: n = 16, N = 4. C9-3: n = 36, N = 4. ∗∗∗ p < 0.001, t test.
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    Waveform rescue and impairment of AIS in C9-MSNs (A) Example AP waveforms for Con-1 (blue) and C9-3 MSNs (black) before and after the addition of BK channel activator NS11021 (10 μM, lighter color shade). Scale bars, 20 mV, 10 ms. (B and C) Mean ± SEM AP amplitude, AHP, half-width, threshold, and rheobase of C9-3 MSNs in the presence and absence of NS11021, along with Con-1 MSNs, were examined in parallel. Statistics, ∗ p < 0.05 and ∗∗∗ p < 0.001, from paired t test or t test . Data: con-1: n = 8, N = 3. C9-3: n = 10, N = 4. (D) NS11021 treatment had no effect on AP output in C9-3 MSNs evoked from current stimulation. Con-1 patched in parallel, shown for reference. Data: Con-1: n = 8, N = 3. C9-3: n = 10, N = 4. Statistics, two-way repeated measures ANOVA followed by Tukey’s multiple comparisons test. (E) Images of Con-1 and C9-3 stained with AIS marker ankyrin-G, neuronal marker <t>MAP2,</t> and DAPI. Scale bars, 50 μm. (F) Mean ± SEM AIS length and distance of start of AIS from the cell soma. Data: Con-1: n = 16, N = 4. C9-3: n = 36, N = 4. ∗∗∗ p < 0.001, t test.
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    Proteintech rabbit
    Waveform rescue and impairment of AIS in C9-MSNs (A) Example AP waveforms for Con-1 (blue) and C9-3 MSNs (black) before and after the addition of BK channel activator NS11021 (10 μM, lighter color shade). Scale bars, 20 mV, 10 ms. (B and C) Mean ± SEM AP amplitude, AHP, half-width, threshold, and rheobase of C9-3 MSNs in the presence and absence of NS11021, along with Con-1 MSNs, were examined in parallel. Statistics, ∗ p < 0.05 and ∗∗∗ p < 0.001, from paired t test or t test . Data: con-1: n = 8, N = 3. C9-3: n = 10, N = 4. (D) NS11021 treatment had no effect on AP output in C9-3 MSNs evoked from current stimulation. Con-1 patched in parallel, shown for reference. Data: Con-1: n = 8, N = 3. C9-3: n = 10, N = 4. Statistics, two-way repeated measures ANOVA followed by Tukey’s multiple comparisons test. (E) Images of Con-1 and C9-3 stained with AIS marker ankyrin-G, neuronal marker <t>MAP2,</t> and DAPI. Scale bars, 50 μm. (F) Mean ± SEM AIS length and distance of start of AIS from the cell soma. Data: Con-1: n = 16, N = 4. C9-3: n = 36, N = 4. ∗∗∗ p < 0.001, t test.
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    Image Search Results


    Waveform rescue and impairment of AIS in C9-MSNs (A) Example AP waveforms for Con-1 (blue) and C9-3 MSNs (black) before and after the addition of BK channel activator NS11021 (10 μM, lighter color shade). Scale bars, 20 mV, 10 ms. (B and C) Mean ± SEM AP amplitude, AHP, half-width, threshold, and rheobase of C9-3 MSNs in the presence and absence of NS11021, along with Con-1 MSNs, were examined in parallel. Statistics, ∗ p < 0.05 and ∗∗∗ p < 0.001, from paired t test or t test . Data: con-1: n = 8, N = 3. C9-3: n = 10, N = 4. (D) NS11021 treatment had no effect on AP output in C9-3 MSNs evoked from current stimulation. Con-1 patched in parallel, shown for reference. Data: Con-1: n = 8, N = 3. C9-3: n = 10, N = 4. Statistics, two-way repeated measures ANOVA followed by Tukey’s multiple comparisons test. (E) Images of Con-1 and C9-3 stained with AIS marker ankyrin-G, neuronal marker MAP2, and DAPI. Scale bars, 50 μm. (F) Mean ± SEM AIS length and distance of start of AIS from the cell soma. Data: Con-1: n = 16, N = 4. C9-3: n = 36, N = 4. ∗∗∗ p < 0.001, t test.

    Journal: Cell Reports

    Article Title: Striatal neuron dysfunction in C9ORF72-FTD/ALS is driven by AIS and potassium channel dysregulation

    doi: 10.1016/j.celrep.2026.117672

    Figure Lengend Snippet: Waveform rescue and impairment of AIS in C9-MSNs (A) Example AP waveforms for Con-1 (blue) and C9-3 MSNs (black) before and after the addition of BK channel activator NS11021 (10 μM, lighter color shade). Scale bars, 20 mV, 10 ms. (B and C) Mean ± SEM AP amplitude, AHP, half-width, threshold, and rheobase of C9-3 MSNs in the presence and absence of NS11021, along with Con-1 MSNs, were examined in parallel. Statistics, ∗ p < 0.05 and ∗∗∗ p < 0.001, from paired t test or t test . Data: con-1: n = 8, N = 3. C9-3: n = 10, N = 4. (D) NS11021 treatment had no effect on AP output in C9-3 MSNs evoked from current stimulation. Con-1 patched in parallel, shown for reference. Data: Con-1: n = 8, N = 3. C9-3: n = 10, N = 4. Statistics, two-way repeated measures ANOVA followed by Tukey’s multiple comparisons test. (E) Images of Con-1 and C9-3 stained with AIS marker ankyrin-G, neuronal marker MAP2, and DAPI. Scale bars, 50 μm. (F) Mean ± SEM AIS length and distance of start of AIS from the cell soma. Data: Con-1: n = 16, N = 4. C9-3: n = 36, N = 4. ∗∗∗ p < 0.001, t test.

    Article Snippet: MAP2 anti-Rabbit (1:1000) , Synaptic Systems , Cat# 188 003; RRID AB_2281442.

    Techniques: Staining, Marker